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Calcium binds to the ryanodine receptor on the sarcoplasmic reticulum which then causes vast release of more calcium. This calcium then forms cross-bridges by binding to the troponin.
Calcium binds to tropinin-C to induce a conformational change which moves tropinin-I away from actin/tropomyosin. Tropomyosin then moves to clear the myosin binding site on the actin thus allowing cross bridge attachment. Hence, contraction is critically dependent on intracellular calcium concentrations.
The process of cross-bridges formation and the shortening of the individual cardiomyocyte require ATP (phosphate group)...
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