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This translocation results in the transfer of the Abelson (ABL) on chromosome 9 oncogene to an area of chromosome 22 termed the breakpoint cluster region (BCR). The resulting hybrid gene ABL-BCR encodes for a protein of p210 or p185 weight from the tyrosine-kinase group. A tyrosine-kinase activates signal pathways, leading to the uncontrolled cell growth associated with this type of cancer. Specifically, the protein interacts with a 3beta(c) receptor subunit, activating the cell cycle and speeding up cell division. CML then proceeds through 3 phases: Chronic, accelerated, and blast crisis. It is with the chronic phase that we are concerned...
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